From 24da0de92836140814da540e5649c11140a14a33 Mon Sep 17 00:00:00 2001 From: sergioclaypool Date: Sat, 4 Oct 2025 17:36:47 +0000 Subject: [PATCH] Add 24.1 Characteristics Of Fungi --- 24.1-Characteristics-Of-Fungi.md | 1 + 1 file changed, 1 insertion(+) create mode 100644 24.1-Characteristics-Of-Fungi.md diff --git a/24.1-Characteristics-Of-Fungi.md b/24.1-Characteristics-Of-Fungi.md new file mode 100644 index 0000000..0e8a2d0 --- /dev/null +++ b/24.1-Characteristics-Of-Fungi.md @@ -0,0 +1 @@ +
Many parasitic fungi possess haustoria, as these constructions penetrate the tissues of the host, release digestive enzymes inside the host's body, and absorb the digested nutrients. Fungi reproduce sexually and/or asexually. Some fungi reproduce each sexually and asexually, while other fungi reproduce only asexually (by mitosis). In both sexual and asexual reproduction, fungi produce spores that disperse from the mother or father organism by either floating on the wind or hitching a trip on an animal. Fungal spores are smaller and lighter than plant seeds. For example, the giant puffball mushroom bursts open and releases trillions of spores in a massive cloud of what looks like finely particulate mud. Fungi reproduce asexually by fragmentation, budding, or producing spores. Fragments of hyphae can grow new colonies. Somatic cells in yeast kind buds. The commonest mode of asexual reproduction is thru the formation of asexual spores, that are produced by a single individual thallus (by means of mitosis) and are genetically an identical to the guardian thallus (Figure 24.8). Spores enable fungi to develop their distribution and colonize new environments.

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Experiments were carried out where nerves were exposed to aglycaemia and the CAP recorded until it began to fall, an indication that glycogen was depleted. The CAP was allowed to get better to its baseline worth after which aglycemia was introduced again, the idea being that the glycogen is not going to be replenished to its baseline level. The latency to CAP failure throughout this second interval of aglycemia was shortened compared to the first interval of aglycemia (Brown et al., 2003). The position of glycogen under more physiological situations was investigated. Within the presence of 2 mM glucose, which is taken into account to be hypoglycemic and a systemic focus that's reached in sort 1 diabetic patients who mismatch insulin supply with prevailing glucose levels, the CAP is maintained for extended intervals of time. However depleting glucose by imposing a interval of aglycemia and then reintroducing 2 mM glucose led to CAP failure, indicating that by itself 2 mM glucose just isn't adequate to assist the CAP, but is supplemented by the breakdown of glycogen to offer supplemental vitality substrate (Brown et al., 2003). Removing that supply of glycogen-derived substrate results in CAP failure, indicating that in kind 1 diabetic patients, throughout intervals of hypoglycemia, glycogen is damaged down to provide supplemental substrate to help mind function.

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